Preliminary reports reveal conolidine may well inhibit certain ion channels, reducing neuronal excitability and limiting soreness alerts. This mechanism is especially applicable in neuropathic agony, wherever irregular signaling results in persistent soreness. Moreover, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to ache notion. Computational docking... https://georgesl214ifa5.wikinewspaper.com/user